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The Gynecologic Oncology Program takes a team approach to patient care. Your team includes gynecologic oncologists, gynecologic oncology surgeons, medical oncologists, radiation oncologists, pathologists, radiologists and oncology nurses. Together, they discuss the diagnosis and staging of the disease and treatment recommendations for every patient. This approach ensures patients receive the benefit of multiple opinions and team input. The diagnosis and treatment of gynecologic cancers have become highly personalized based on genetic markers, family history and other factors unique to you.
Treatment options for gynecologic cancers include:. Genetic susceptibility to HPV infection and cervical cancer. Brazilian Journal of Medical and Biological Research. Genetic susceptibility of cervical cancer. Journal of Biomedical Research. Loss of heterozygosity analyzed by single nucleotide polymorphism array in cancer. World Journal of Gastroenterology. Loss of heterozygosity on chromosome 6p Clinical cancer research: an official journal of the American Association for Cancer Research. Comprehensive analysis of loss of heterozygosity events in glioblastoma using the K SNP mapping arrays and comparison with copy number abnormalities defined by BAC array comparative genomic hybridization.
World journal of gastroenterology: WJG. Loss of heterozygosity as a predictor to map tumor suppressor genes in cancer: molecular basis of its occurrence. Current opinion in oncology. Hunt JL. In: ed.
Stoler MH. Cell and Tissue Based Molecular Pathology. Philadelphia: Churchill Livingstone. Genome-wide association study of susceptibility loci for cervical cancer. Molecular biology techniques for loss of heterozygosity detection: the glioma example. Jornal Brasileiro de Patologia e Medicina Laboratorial. Heritability of cervical tumours. Ivansson E. Contribution of Immunogenetic factors in susceptibility to cervical cancer [PhD Dissertation].
Uppsala University: Uppsala University.
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Factors contributing to risk for cancer among HIV-infected individuals, and evidence that earlier cART will alter this risk. Genetic polymorphisms as predictive and prognostic biomarkers in gynecological cancers. A genome-wide association study identifies two new cervical cancer susceptibility loci at 4q12 and 17q Nat Genet. Garner D. Clinical application of DNA ploidy to cervical cancer screening: A review. World Journal of Clinical Oncology.
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The presence of extra chromosomes leads to genomic instability. Nature communications. Revisiting tumour aneuploidy — the place of ploidy assessment in the molecular era. Nature Reviews Clinical Oncology. Barh D, Gunduz M. Integrative genomics analysis of chromosome 5p gain in cervical cancer reveals target over-expressed genes, including Drosha.
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Molecular Cancer. Small supernumerary marker chromosomes and their correlation with specific syndromes. Advanced Biomedical Research. Characterization of genomic changes in the cervical pre-cancerous lesions and tumors induced by different types of human papillomaviruses. TP53 codon 72 polymorphism and cervical cancer: a pooled analysis of individual data from 49 studies.
The Lancet Oncology. Combined array-comparative genomic hybridization and single-nucleotide polymorphism-loss of heterozygosity analysis reveals complex genetic alterations in cervical cancer.
BMC Genomics. Signatures of mutational processes in human cancer. Immuno-related polymorphisms and cervical cancer risk. The IARC multicentric case-control study. Public Library of Science. Genetic variation in CD83 and risks of cervical and vulvar cancers: a population-based case-control study. CD83 polymorphisms and cervical cancer risk.
Kim YT, Zhao M. Yonsei Med J. Genetic susceptibility in cervical cancer: From bench to bedside. Journal of Cellular Physiology. Journal of Assisted Reproduction and Genetics. Clinical Cancer Research. Analysis of the genetic architecture of susceptibility to cervical cancer indicates that common SNPs explain a large proportion of the heritability. Mathew CG. Fanconi anaemia genes and susceptibility to cancer. High incidence of female reproductive tract cancers in FA-deficient HPVtransgenic mice correlates with E7's induction of DNA damage response, an activity mediated by E7's inactivation of pocket proteins.
Differences in genetic variation in antigen-processing machinery components and association with cervical carcinoma risk in two Indonesian populations. Molecular backgrounds of ERAP1 downregulation in cervical carcinoma. Analytical Cellular Pathology. CD gene variant and susceptibility to cervical cancer: a meta analysis. International Journal of Clinical and Experimental Medicine. Gynecologic and Obstetric Investigation. Pathway analysis of cervical cancer genome-wide association study highlights the MHC region and pathways involved in response to infection. Pooled analysis of genome-wide association studies of cervical intraepithelial neoplasia 3 CIN3 identifies a new susceptibility locus.
International journal of women's health. Cancer Epidemiology and Prevention Biomarkers. Nature genetics.
Cytotoxic T-lymphocyte-associated antigen-4 polymorphisms and susceptibility to cervical cancer: A meta-analysis. Molecular medicine reports. Predictive genetic testing in children: constitutional mismatch repair deficiency cancer predisposing syndrome. Journal of genetic counseling.
Communicating cancer risk within an African context: experiences, disclosure patterns and uptake rates following genetic testing for Lynch syndrome. Patient education and counseling. CTLA-4 gene and susceptibility to human papillomavirusassociated cervical squamous cell carcinoma in Taiwanese women. Evasion of host immune defenses by human papillomavirus. Common genetic variants and risk for HPV persistence and progression to cervical cancer. The role of p73 G4Cto-A4T14 polymorphism in the susceptibility to cervical cancer. DNA and cell biology.
Peroxiredoxin 3 is a novel marker for cell proliferation in cervical cancer. Biomedical Reports. Kardani K, Bolhassani A. Role of inflammasome genetics in susceptibility to HPV infection and cervical cancer development. Journal of medical virology. Common variants in immune and DNA repair genes and risk for human papillomavirus persistence and progression to cervical cancer.
Cell reports. Gene network reconstruction reveals cell cycle and antiviral genes as major drivers of cervical cancer. Ganguly N, Parihar SP. Human papillomavirus E6 and E7 oncoproteins as risk factors for tumorigenesis.
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Journal of biosciences. Polymorphism of the pre-miRa is associated with risk of cervical cancer in a Chinese population. BioMed Research International. J Biol Chem. Human papillomavirus and host genetic polymorphisms in carcinogenesis: A systematic review and meta-analysis. URG4 overexpression is correlated with cervical cancer progression and poor prognosis in patients with early-stage cervical cancer.
Scientific Reports. The American Journal of Pathology.
F.R.E.E [D.O.W.N.L.O.A.D] Noninvasive Molecular Markers in Gynecologic Cancers by
Journal of virology. Current Genomics. Polymorphism of CYP1A1 gene variants rs and rs relation to the incidence of cervical cancer in Chhattisgarh. Save this study. Warning You have reached the maximum number of saved studies Listing a study does not mean it has been evaluated by the U. Federal Government. Read our disclaimer for details. Last Update Posted : December 23, Study Description. The purpose of this prospective multicenter trial is to investigate the value of molecular markers in endometrial cancer for predicting lymph node metastasis and prognosis in relation to treatment.
This is a prospective multicenter study to investigate the predictive value of molecular markers in endometrial cancer for lymph node metastasis, prognosis and treatment. For the previously studied tumor markers p53, p16, ER, PR and HER2neu, we want to investigate the expression in curettage material in relation to lymph node metastasis and prognosis among endometrial carcinoma patients. We also want to investigate the distribution of genetic alterations in fresh frozen tumor tissue in order to design prospective randomized treatment trials of metastatic endometrial cancer based on molecular profile.
Tumor specimens from endometrial cancer patients, collected preoperatively and during primary hysterectomy, are investigated. Outcome Measures. Eligibility Criteria. Information from the National Library of Medicine Choosing to participate in a study is an important personal decision. Women with endometrial carcinoma that undergo an endometrial biopsy before treatment with hysterectomy with bilateral salpingoophorectomy with or without pelvic lymph node staging.
Inclusion Criteria: Women with endometrial carcinoma Available endometrial biopsy Informed consent Exclusion Criteria: No informed consent. Contacts and Locations. Information from the National Library of Medicine To learn more about this study, you or your doctor may contact the study research staff using the contact information provided by the sponsor. Please refer to this study by its ClinicalTrials.
Layout table for investigator information Principal Investigator: Helga B. Salvesen, Prof.